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Further, our analyses revealed that the multi-omics signature associated with higher circulating TMAO concentrations and, therefore, elevated cardiovascular risk, shifted from obesity with increased insulin resistance toward overt T2D and IHD with only three variables (age, eGFR, and serum butyryl-carnitine) consistently being strong contributors across models built with the BMIS, T2D and IHD disease groups

Ehlers-Danlos Syndrome : Physical therapy, joint stabilization, and pain management are crucial for EDS patients with POTS

DNA sequencing identified two putative null mutants, Gmmorf2a/b-1 and Gmmorf2a/b-2 , which harbored non multiple of 3-bp deletions in the first exon of GmMORF2a and GmMORF2b , causing frameshifts and premature termination of protein translation (Supplementary Fig

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an exaggerated appearance of gastric lesions was noted in control diabetic rats