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[1] [2] The medication works by binding to GLP-1 receptors in pancreatic beta cells, enhancing glucose-dependent insulin secretion, suppressing glucagon release, and slowing gastric emptying
Direct GLP-1 activation evidence is limited
This is particularly effective when insulin resistance is the primary driver of elevated VLDL
If someone on a GLP-1 drug isnt eating enough food namely, carbohydrates blood sugar can drop too quickly
Both NNC0090-2746 (107) and tirzepatide (63) delivered clinically meaningful improvements in glycemic control and body weight reduction in their phase 1 trials
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