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Diabetes Care 39 , 222230 (2016)

Consequently, nanotechnology researchers are exploring innovative strategies to modulate checkpoint pathways in tumor models, aiming to develop approaches that are both safer and more broadly effective [44]

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Excessive generation of ROS within ovarian, adipose, and hepatic tissues overpowers antioxidant defenses, leading to oxidative damage of lipids, proteins, and mitochondrial DNA ( Androgen excess exacerbates redox disequilibrium by promoting NADPH oxidase activity and mitochondrial ROS production, while simultaneously impairing follicular maturation and oocyte quality ( Figure 1 represents the involvement of redox imbalance and endocrine dysregulation in the ovary, leading to hyperandrogenism or PCOS