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Crucially, wild-type MZB are known to be prone to activation compared to FoB 7,8,75 , and we found that both Gclc fl/fl Mb1-Cre + FoB and B6 MZB showed a similar increase in mitochondrial mass (Fig

Therapeutics based on mitochondrial dysfunction and oxidative stress, such as mitochondria-targeted antioxidant [10-(4,5-dimethoxy-2-methyl-3,6-dioxo-1,4-cyclohexadien-1-yl)decyl]triphenylphosphonium methanesulfonate (MitoQ), modify disease progression by slowing the decline of mitochondrial function and disease progression in the SOD1 G93A mouse model of ALS (Miquel et al., 2014)

However, heart disease is multifactorial

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