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Indeed, mitochondrial impairment caused by mutations in genes linked to familial PD, together with data from human post-mortem tissue indicate that impaired mitochondrial function, increased oxidative stress and deficient anti-oxidant capacity are common pathological mechanisms implicated in the etiology of both familial and sporadic PD cases (Dauer and Przedborski, 2003
Stimulation of collagen synthesis in fibroblast cultures by the tripeptide-copper complex GHK. FEBS Letters , 238(2), 343346
The pericytea review
The end result wrinkles and sagging skin
doi:10.1016/j.jad.2010.12.019, PMID: 21277635 Maes M, Van de Vyvere J, Vandoolaeghe E, Bril T, Demedts P, Wauters A, Neels H (1996) Alterations in iron metabolism and the erythron in major depression: further evidence for a chronic inflammatory process
