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Recent research suggests that modulating autophagic flux could be a potential therapeutic strategy for various cardiovascular conditions, including atherosclerosis, coronary artery disease, diabetic cardiomyopathy, arrhythmia, chemotherapy-induced cardiotoxicity, and heart failure [106]
28 For example, p70S6K1 phosphorylates MDM2 at Ser166 resulting in polyubiquitination and degradation of p53 and thereby inhibits p53-dependent apoptosis
However, the lack of clinical trials means that there could potentially be serious BPC-157 side effects in humans, which remain unknown
They are no longer dendritic
