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Suppression of human immunodeficiency virus expression in chronically infected monocytic cells by glutathione, glutathione ester, and N-acetylcysteine
First, overexpression of the alsin long isoform protects against motor neuron toxicity induced by expression of A4T, or mutant SOD1 G85R or SOD1 G93A (Kanekura et al., 2004)
N-acetylcysteine augmentation to tranylcypromine in treatment-resistant major depression
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