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The pathological role of advanced glycation end products-downregulated heat shock protein 60 in islet -cell hypertrophy and dysfunction
[3] While a theoretical mechanism occurs by which exposure to severe stressors and high levels of subsequent distress such as post-traumatic stress disorder could increase the risk of immune disease and cause an aggravation of the autoimmune response that leads to Graves disease, more robust clinical data are needed for a firm conclusion
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In support of this hypothesis, we found that GCLC protein and mRNA exhibit an inverse correlation with MYC signaling in vivo
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