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5 September 2017
Spring Harb
Transgenic AD models such as APP/PS1 mice, which overexpress mutant human amyloid precursor protein and presenilin-1, have also revealed mitochondrial fragmentation and respiratory deficits, reinforcing the hypothesis that impaired mitochondrial dynamics and mitophagy are mechanistically linked to AD pathology
People who take GLP-1 weight-loss medications and then stop them are likely to experience strong metabolic rebound, including rapid weight regain and worsening blood pressure and blood sugar levels
Causes of Peripheral Neuropathy
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