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We examine its modulation of mitochondrial function, oxidative stress, inflammation, autophagy, and apoptosis across major cardiac conditions, including ischemia/reperfusion injury, heart failure, diabetic cardiomyopathy, and anthracycline-induced cardiotoxicity

10.1038/s41598-020-71882-2 55 IntakhanN.SaeungA.Rodrigues OliveiraS

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However, under hypoxic conditions, HIF-1 inhibits ferritinophagy flux and protects osteoclasts from ferroptosis by inhibiting the formation of autophagosomes