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[1] No specific antibiotics are contraindicated for concurrent use with semaglutide in the FDA prescribing information
In the context of concurrent GLP-1 agonism which is hypothesised to counterbalance glucagon's hyperglycaemic effects net glucagon receptor stimulation may enhance fat metabolism without causing clinically significant hyperglycaemia
A variety of approaches have been evaluated but with limited success, including stem cells, growth factors, cytokine modulators, antimicrobial and anti-inflammatory drugs, matrix metalloproteinase regulators, angiogenic stimulators, extracellular matrix promoters, and several phytopharmaceuticals
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