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The transport of drug molecules from the nasal cavity to the brain involves two primary pathways: intracellular and extracellular

Animal Model Evidence The systemic evidence base is entirely preclinical: Pulmonary fibrosis (mouse model): Intraperitoneal GHK-Cu at 0.2-20 micrograms/g/day protected against bleomycin-induced lung fibrosis by inhibiting TGF-beta1/Smad signaling and reducing oxidative stress (PMID 31809714)

Characterisation of nanoplastics during the degradation of polystyrene

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Others might theoretically conflict with metabolic pathways already being modulated