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The experiments involved 6- to 12-week-old male mice with a C57BL/6J genetic background
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replacing or modulating those signals can be very powerful
Serious risks can include pancreatitis, gallbladder disease, kidney injury related to dehydration, allergic reactions, and worsening diabetic retinopathy in some people with diabetes eye disease
Evaluation of long acting GLP1R/GCGR agonist in a DIO and biopsy-confirmed mouse model of NASH suggest a beneficial role of GLP-1/glucagon agonism in NASH patients
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