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TrxR1 is predominantly a cytoplasmic enzyme [52], while TrxR2 is mitochondrial [53]
This truncation substantially reduces IGFBP binding affinity while preserving high-affinity engagement with the IGF-1 receptor (IGF1R), resulting in greater free peptide availability and potentially enhanced receptor signaling per unit of administered peptide in research models
Nutritional quality becomes paramount
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Balancing Weight and Muscle Loss in GLP1 Receptor Agonist Therapy. Nature Reviews Endocrinology , 28 July 2025
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