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As mentioned above, ferroptosis could be induced to exert anti-tumor functions via signaling pathway modulation, non-coding RNA expression, DNA methylation, histone modification, CSCs, microenvironment remodeling, and so on

In animal models of ulcers, colitis, and intestinal permeability (leaky gut), it supports epithelial regeneration, stabilizes mucosal barriers, and maintains gastrointestinal integrityhighlighting its broad systemic potential

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During remodeling, they support proper organization and functional repair

Without glutathione, the brain cannot handle free radicals as well, and more damage occurs