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The expression profile of growth factors involved in OPC recruitment (PDGFRa, FGF2) and in OPC differentiation (IGF1, TGF1) following demyelination is altered in old mice, in which the upregulation of these factors is both weaker and delayed (Hinks and Franklin, 2000)

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However, studies have shown that antioxidant approaches, including anti-neuroinflammatory and neuroprotective agents, may have therapeutic efficacy in the treatment of PD ( The interplay between mitochondrial function and GSH is especially critical in PD