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Meanwhile, IL-1 upregulates IFI16 expression via m6A epigenetic modification, inducing inflammatory apoptosis of PAECs, revealing the epigenetic regulation of metabolic pathways by inflammatory factors (Rao et al., 2025)
In a similar way, in an MMAA, the NOP receptor agonist UFP-112 , administered in OVA-sensitized mice, reduced eosinophilic infiltration and T cell proliferation, with a decrease in the Th2 profile and increased IFN- levels, effects that were blocked by the antagonist UFP-101 (Sullo et al., 2013)
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