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The development of rheumatoid arthritis is based on a genetic predisposition, such as the HLA-DR4 alleles, that create antibodies against citrullinated proteins, and on environmental triggers such as infection or smoking [8]

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In contrast, good fits with concentration-independent rate constants were obtained for the monothiol mechanism (iii) from Fig

Targeted metabolomics analysis shows that 5FU treatment promotes significant changes in metabolite abundance over time (Fig