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Within experimental models, glucagon receptor activation has been associated with: Hepatic fatty acid oxidation Lipid mobilization Changes in substrate utilization Increased energy expenditure Brown adipose tissue activation These pathways have become an area of considerable scientific interest because they extend beyond the traditional mechanisms targeted by earlier incretin therapies
Thyroid hormone and electrolyte levels may be measured as abnormal levels are associated with increased risk of arrhythmia (NHLBI 2011b)
[Medline]
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These biomarkers are also used to track the efficacy of antioxidant therapies, with decreasing levels of MDA and 4-HNE indicating effective intervention [35]
