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10.1016/j.biopsych.2011.03.028 89 CosorichI.Dalla-CostaG.SoriniC.FerrareseR.MessinaM

Mitochondrial dysfunction in ASD is further supported by the presence of non-specific biomarkers including reduction in carnitine and increases in lactate, pyruvate, creatine kinase, aspartate aminotransferase and alanine aminotransferase compared to control groups (Rossignol and Frye, 2012)

In combination with other agents, it can cause gene expression changes that single agents do not

Secondary carnitine deficiencies are most commonly the result of trapping of carnitine in fatty acylcarnitine due to defects in mitochondrial fatty acid -oxidation enzymes such as is the case in medium-chain acyl-CoA dehydrogenase deficiency (MCADD)