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Studies have found that GHK-Cu increases integrin expression and the transcription factor p63, both of which support the proliferative and synthetic activity of dermal fibroblasts
In preclinical rodent research it is characterized as a cytoprotective and angiomodulatory agent, reported to influence the VEGFnitric-oxide signaling axis, modulate angiogenesis during muscle and tendon healing, and promote granulation-tissue and collagen formation at wound sites
Topical GHK-Cu human trials and systemic animal studies reveal an excellent tolerability profile with no documented toxicity
The researchers also suggested that The enhancement of wound closure was more pronounced (median of 89.2% compared with -10.3% for vehicle
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