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Available evidence suggests that AMY2R and the calcitonin receptor are both expressed in osteoclasts [10-12] , indicating that amylin and calcitonin receptor agonism may exert predominantly anti-resorptive effects

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While GHK has been shown to inhibit the inflammatory response and epithelial-mesenchymal transition (EMT) via the TGF-1/Smad 2/3 and IGF-1 pathways in bleomycin-induced fibrosis models [24], its broader anti-inflammatory effects in the context of pulmonary fibrosis pathogenesis may warrant further exploration

Contribution statement AK and SAA contributed to the concept of the study and study design

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