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The liver expresses IGF1R alongside its dominant role in producing IGF-1, and autocrine/paracrine IGF1R signaling contributes to hepatocyte biology in research models
Now, as for the reversibility of nerve damage: In terms of neurological recovery from B12 deficiency, studies conducted on rats so far indicate that methyl B12 may promote the regeneration of nerves: Schwann cells (SCs) are constituents of the peripheral nervous system
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