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The origin of CAFs remains unclear, and some propose that they derive from the activation of resident fibroblast [98, 99], from recruited bone marrow progenitors [100], or the transdifferentiation from endothelial [101, 102] or epithelial to mesenchymal [103]
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In light of these challenges, in this study, we assess the impact of GLP-1 and its derivatives on pain behavior and their influence on the peripheral nervous system
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