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PT-141 acts as a melanocortin receptor agonist, primarily active at the MC3R and MC4R receptors in the hypothalamus
Conditions such as metabolic stress, acute or chronic inflammation or nutritional availability all might impact the regulation of GLP-1 from intestinal or pancreatic sources [13]
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This allows them to actively manage any side effects, which may subside by bedtime
Another preclinical study in mice had determined that another GLP-1RA, exendin-4, reduced the development of atherosclerosis and decreased the number of monocytes located on the vascular endothelium, further highlighting the anti-inflammatory role of GLP-1 [46]
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