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Inflammatory bowel disease (IBD) model: Relieve inflammation, repair barriers A mouse ulcerative colitis (UC) model induced by DSS (dextran sulfate sodium) was used to simulate intestinal inflammation and barrier damage in human IBD, and the protective effect of IGF-1 LR3 was studied

Although the drop in hepatic or renal GSH is the most toxicologically relevant interaction (see also below), plasma GSH, free cysteine (Nuttall et al., 2003) were significantly reduced after a single 2 g PAC administration or 14 days of therapeutic doses of PAC in human volunteers, respectively