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Redox Resilience: How Glutathione and Mitochondrial Function Shape Cardiovascular Risk Oxidative stress and mitochondrial dysfunction are increasingly recognized as central drivers of cardiovascular disease, influencing endothelial function, inflammation, lipid oxidation, and metabolic resilience

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95% CI, 7.45% to 29.11%

In ageing septic rats, ghrelin/growth hormone treatment reverses immunosuppression by inhibiting the production of TGF- through the vagus nerve (Zhou et al