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Confirming the important role of the TGF/SMAD pathway in pancreatic fibrosis, we found that high glucose promoted the activation of PSCs in vitro

As a consequence, the unrestrained and potentially lethal accumulation of ROS threatens to damage many biomolecules: lipids, proteins, DNA, and host cells

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Nitric oxide synthase pathway engagement represents another primary mechanism of BPC-157 action

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