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Figure 2 2.1 mtDAMPs release and innate immune activation 2.1.1 Tissue injury and mitochondrial danger signals During surgery and acute trauma, abrupt tissue damage and hypoxia lead to cellular necrosis, releasing not only cytosolic contents but also mitochondrial constituents into the circulation
Heterozygote ornithine transcarbamylase deficiency presenting as symptomatic hyperammonemia during initiation of valproate therapy
Diphenyl diselenide administration enhances cortical mitochondrial number and activity by increasing hemeoxygenase type 1 content in a methylmercury-induced neurotoxicity mouse model
2020 jan:151:104554)
H 2 O, Milli-Q, and IS: Kynurenine-D6 and Tryptophan-D8 were obtained from Sigma-Aldrich (St
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