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Funding This paper was supported by San Matteo Hospital Foundation, Internal Medicine research fundings, PRIN2017. Publishers note All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers
Since NMN (nicotinamide mononucleotide) was previously reported to increase the levels of NAD + by acting as the precursor for NAD + 27 , we investigated whether NMN treatment would have therapeutic impact on diseased UMNs
The PDHC-catalyzed reaction generates not only NADH, which is oxidized in the mitochondrial respiratory chain, but also acetyl-CoA, which feeds into the TCA cycle and is a precursor of acetylcholine (ACh) in cholinergic cells
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