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Pg , Fn , and Sm groups exhibited a downregulation in FMO2 gene expression compared to the control group, which potentially indicates a general stress response of the liver to oral pathogenic bacterial infection ( In vitro TMAO exacerbates lipid accumulation, inflammation, and fibrosis in the liver ( in vitro ( Pg orally exhibited hepatic steatosis, accompanied by increased expression levels of lipid metabolism factors

Beyond its safety and accessibility, delgocitinibs broad but targeted mechanism of action, which targets multiple JAK pathways downstream of various cytokines, enables it to address the diverse immune dysregulation seen in CHE [58, 106]

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