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their accumulation at presynaptic terminals and postsynaptic dendritic spines is increased when the synaptic activity is elevated [31]

repletion reverses hematologic symptoms within days and neurologic sequelae over weeks, reflecting differential tissue turnover

Mounting evidence indicates that the pharmacological inhibition of ferroptosis exerts neuroprotective effects in vitro and in vivo epileptic disease models (Table 1

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