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In this particular, deleterious mechanisms of brain damage often associated with and usually consequent to excitotoxicity, such as oxidative stress and bioenergetic disruption elicited by the accumulated metabolites, were prevented by blocking the overstimulation of NMDAR that leads to a high influx of calcium into the cells and are followed by increased production of ROS and impairment of electron transport chain (ETC) [121]

FASEB journal : official publication of the Federation of American Societies for Experimental Biology , 34 (2), 32893304