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The original ipamorelin paper showed a GHRP-like receptor response using GHRP and GHRH antagonists, but it did not prove that ipamorelin has a unique receptor conformation, a unique signaling pathway, or a fully characterized biased-agonism profile

Interesting, I didn't realize how connected those two really are

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3A), hence supporting our hypothesis that RSL3-induced PTC cell death is indeed a ferroptosis pathway-dependent phenomenon