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Microbiota-driven ACh dysregulation is validated in inflammatory bowel disease (IBD) models: Dysbiosis-induced choline deficiency is reversed by cytidine diphosphate (CDP)-choline supplementation, which upregulates choline transporter (ChT1), acetylcholinesterase (AChE), and 7 nicotinic acetylcholine receptor (7nAChR) expression to restore neurotransmission (Guo et al., 2023a)

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S-nitrosoglutathione as a substrate for -glutamyl transpeptidase