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65,66 Notably, molecular changes exhibited by non-proliferative reactive astrocytes can be fully reversible over time after single exposures for example to neuroinflammation caused by LPS
Not having enough intrinsic factor, a stomach protein needed for B12 absorption
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Keywords , metabolic dysfunction-associated steatohepatitis , incretin therapy , glucagon-like peptide-1 , glucose-dependent insulinotropic polypeptide , glucagon receptor agonism 0 1 INTRODUCTION Metabolic dysfunction-associated steatotic liver disease (MASLD) is defined by hepatic steatosis, characterized by triglyceride accumulation in more than 5% of hepatocytes, alongside any cardiometabolic risk factor establishes a diagnosis of MASLD [1]
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