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Regular monitoring continues for one to two years after brace discontinuation to ensure the curve remains stable.

A lutein nanoassembly (lutein@DTPP) constructed based on DSPE-thioketal-PEG2K enables myocardial targeting and ROS-responsive drug release, improving mitochondrial function by regulating the MDM2-NDUFS1 axis and inhibiting ferroptosis in myocardial cells during myocardial ischemia-reperfusion injury (152)

Injectable BPC-157 is preferred for musculoskeletal injuries, as it allows targeted delivery to the affected area and higher systemic bioavailability

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