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Triple-action cancer therapy using laser-activated NO-releasing metallomicellar nanophotosensitizer for pyroptosis-driven immune reprogramming
The peptide activates endothelial nitric oxide synthase (eNOS) through both VEGFR2-dependent and VEGFR2-independent pathways
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mitigated by the GLP-1 component Safety database : Primarily from Chinese Phase 3 trials
NO, as a lipophilic molecule, easily diffuses from endothelium into adjacent vascular smooth muscle cells (VSMCs) and binds to the prosthetic haem group of soluble guanylate cyclase (sGC), thus activating the enzyme [7]
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