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It is given to treat pernicious anemia, cyanide poisoning, Leber's optic atrophy, and toxic amblyopia
New patients require an initial consultation
Activation of the Nrf2 response by intrinsic hepatotoxic drugs correlates with suppression of NF-\(\kappa\)B activation and sensitizes toward TNF\(\alpha\)-induced cytotoxicity
In conclusion, the present work provides crucial biosynthetic insights for a more sustainable production of designer anthocyanins ( 1 ) and is anticipated to drive the construction of powerful, industrial anthocyanin-producing strains, further fuelling the ongoing biologization of the food and nutraceutical sectors
This observation necessitates the existence of LDHA-independent anti-ferroptosis mechanisms
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