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Without a spatially restricted and experimentally identifiable origin of ferroptotic stress, ordered patterns of cell death could simply reflect pre-existing gradients in ferroptosis susceptibility within heterogeneous microenvironments rather than true directional propagation
FOXO4-DRI interrupts the interaction between FOXO4 and p53, destabilizing the survival mechanisms of senescent cells
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Glutathione may interact with certain medications, which include chemotherapy drugs and antioxidant supplements
To resolve the complex crosstalk among these pathways, machine-learning frameworks built on PANoptosis-related risk scores (PRS) comprised of six key hub genes (YWHAH, PRKACB, PSMB9, FAS, GZMA, and CASP1) have revealed extensive molecular co-activation within tubular and interstitial segments, strongly predicting eGFR decline, serum creatinine elevation, and local immune cell recruitment (209)
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