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H., and Trimmer, G
Moharamzadeh K, Brook IM, VanNoort R, Scutt AM, Thornhill MH
Metabolomics and lipidomics can identify substrate stress, mitochondrial bottlenecks, amino acid imbalance, redox dysregulation, and systemic drivers such as diabetes or chronic kidney disease ( This review synthesizes recent evidence on key metabolic derangementssubstrate inflexibility, mitochondrial dysfunction, BCAA accumulation, and ketone adaptationin myocardial hypertrophy, and critically examines their potential as clinically interpretable biomarker modules and therapeutic targets, while highlighting the persistent challenges in distinguishing adaptive from maladaptive remodeling and translating pathway signals into practice
f , Venn diagram showing the overlap between the SynGO database and the 4042 differentially expressed genes in response to GLP-1MK-801 relative to semaglutide
