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Compared with free Cu 2+ , the more membrane damage caused by ATA@Cu NPs may be attributed to the adhesion of ATA@Cu NPs onto MRSA and MRPA with local release of high concentrations of Cu 2+
These findings reposition bone as an active endocrine organ rather than a passive target of hormonal regulation
Three months later a commentary by different Authors hypothesized that NSAIDs could also increase the risk of thrombosis, acute respiratory distress syndrome (ARDS), and acute renal failure in COVID-19 patients (Cumhur Cure et al., 2020)
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