Phosphorylated full-length BH3-interacting domain death agonist (FL-Bid) reversibly increases apoptosis initiators during mitosis, and Bid phosphorylation during mitosis-induced apoptosis initiation depends on voltage-dependent anion-selective channel protein 2 (VDAC2), suggesting that the Bcl2 family interaction network plays a crucial role in regulating the timing of apoptosis initiation [32]
Monomeric C-reactive protein affects cell injury and apoptosis through activation of p38 mitogen-activated protein kinase in human coronary artery endothelial cells
297,299 When IB is phosphorylated and degraded, NF-B can move to the nucleus, activating multiple inflammation-related genes
By comparatively analyzing the overall transcriptional profile of the MC58 strain, belonging to the hyperinvasive cc32 (ST-32), with that of the 710 strain, a carrier isolate belonging to the cc136 (ST-136), during adhesion to human nasopharyngeal cells, Joseph et al
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