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Collectively, these data might indicate that the diminished ROS buffering of GSH-deficient cells leads to an increased activation of the mTOR pathway which could drive to the MZB-like properties of Gclc -deficient FoB

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PT-141 is designed to influence this process at its source by interacting with the melanocortin system within the central nervous system

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Blood 104 , 11911197 (2004)

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