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Through a series of point mutations and protein quantification experiments, it was concluded that Lys-426 (K426) in the C-terminal box is essential for the observed stationary phase-specific degradation of Gdh1p [23]
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Behavioral tests in mice Baseline heat sensitivity was assessed to evaluate the systemic effects of glucose or GLP-1(736) administration via a hot plate apparatus (Ugo Basile, Italy)
The glutathione cycle can compensate for decreased excitatory neurotransmission when the glutamate-glutamine shuttle is inhibited
The satiety effect of glucagon was blocked after disconnection of the hepatic branch of the abdominal vagus nerve [55]
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