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G6PD deficiency is an X-linked enzyme deficiency disorder encompassing numerous allelic variants causing dysfunction of the G6PD enzyme

Among the most widely used mechanisms are reduction-sensitive disulfide (-SS-) and diselenide (-SeSe-) linkages, which can be cleaved by intracellular GSH to activate drug release or imaging functions [16]

The patient also presented with skin hyperextensibility, joint hypermobility, and Gorlin sign (ability to reach the nose with the tip of the tongue) (Fig

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