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2.3.3 Interconnected dynamics of cellular aging: the telomere-mitochondrial axis Telomere shortening can disrupt normal cellular function and is implicated in the increased production of ROS, which further contribute to mitochondrial dysfunction and cell aging (Sahin and DePinho, 2010)

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The lag in formal education can be made good through intensive functional training

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FOXO4-DRI is studied within this context to examine how targeted senolysis influences organ-level structure

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NRF2 regulation processes as a source of potential drug targets against neurodegenerative diseases

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