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Diabetes 48 , 22702276, doi:10.2337/diabetes.48.12.2270 (1999)
Ethanol metabolism, CYP2E1 induction, compromised antioxidant defenses, mitochondrial injury, activation of Kupffer and stellate cells, hypoxia, and iron overload can all contribute to the alcohol-induced oxidative environment
The FDA does not explicitly require a single fixed-dose maintenance strategy
For instance, adverse events (AE) such as sixth cranial nerve palsy, hyperglycemic hyperosmolar nonketotic syndrome, elevated glycated hemoglobin, and positional dizziness are more likely to occur, which are not specifically mentioned in the product labeling
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