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For example, in post-MI patients (n = 78, age 60.2 14.2), addition of L-arginine (3 3 g/d) to the post-MI treatment regimen for 6 months does not improve vascular stiffness or left ventricular function compared to patients receiving placebo (n = 75, age 60.4 12.9) (Schulman et al., 2006)

This decline appears causally related to decreased regenerative capacity, as exogenous administration restores youthful tissue repair patterns

Although current research supports ferroptosis as a central regulator of dopaminergic neuron loss in AD, providing a new mechanistic anchor to link iron accumulation, A aggregation, and tau tangles, key limitations remain