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When nutrient cofactors are insufficient, oxidative demand is excessive, or sulfur metabolism is impaired, glutathione production may fail to keep pace with need
Further analyses of differentially expressed genes between GLP-1MK-801 and semaglutide treatment groups support this notion by showing enrichment of functional terms pertaining to glutamatergic signalling and synaptic plasticity among the most upregulated genes
The excellent clinical manifestations of Tan IIA provide supportive evidence for targeting the GSH system in the treatment of heart diseases, which may be promising for transforming natural compounds from basic research to clinical applications
